Search results for "Nonsynaptic plasticity"

showing 4 items of 4 documents

Dysregulated Expression of Neuregulin-1 by Cortical Pyramidal Neurons Disrupts Synaptic Plasticity

2014

Summary Neuregulin-1 ( NRG1 ) gene variants are associated with increased genetic risk for schizophrenia. It is unclear whether risk haplotypes cause elevated or decreased expression of NRG1 in the brains of schizophrenia patients, given that both findings have been reported from autopsy studies. To study NRG1 functions in vivo, we generated mouse mutants with reduced and elevated NRG1 levels and analyzed the impact on cortical functions. Loss of NRG1 from cortical projection neurons resulted in increased inhibitory neurotransmission, reduced synaptic plasticity, and hypoactivity. Neuronal overexpression of cysteine-rich domain (CRD)-NRG1, the major brain isoform, caused unbalanced excitato…

MaleDendritic SpinesNeuregulin-1Nonsynaptic plasticityGene ExpressionMice TransgenicNeurotransmissionInhibitory postsynaptic potentialSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyCell MovementInterneuronsConditioning Psychologicalmental disordersAnimalsNeuregulin 1lcsh:QH301-705.5CA1 Region HippocampalNeuronal PlasticitybiologyPyramidal CellsAnatomyFearCortex (botany)Synaptic fatiguelcsh:Biology (General)Synaptic plasticitybiology.proteinExcitatory postsynaptic potentialFemaleNerve NetNeuroscience
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Synaptopodin regulates denervation-induced homeostatic synaptic plasticity

2013

Synaptopodin (SP) is a marker and essential component of the spine apparatus (SA), an enigmatic cellular organelle composed of stacked smooth endoplasmic reticulum that has been linked to synaptic plasticity. However, SP/SA-mediated synaptic plasticity remains incompletely understood. To study the role of SP/SA in homeostatic synaptic plasticity we here used denervation-induced synaptic scaling of mouse dentate granule cells as a model system. This form of plasticity is of considerable interest in the context of neurological diseases that are associated with the loss of neurons and subsequent denervation of connected brain regions. In entorhino-hippocampal slice cultures prepared from SP-de…

Patch-Clamp TechniquesDendritic SpinesGreen Fluorescent ProteinsNonsynaptic plasticityMice TransgenicTetrodotoxinBiologyIn Vitro TechniquesHippocampusReceptors N-Methyl-D-AspartateMiceHomeostatic plasticitySynaptic augmentationMetaplasticityAnimalsEntorhinal CortexHomeostasisPromoter Regions GeneticMultidisciplinarySynaptic scalingNeuronal PlasticityMicrofilament ProteinsRyanodine Receptor Calcium Release ChannelBiological SciencesDenervationSpine apparatusMice Inbred C57BLSynaptic fatigueSynaptic plasticityDentate GyrusSynapsesCalcium ChannelsNeuroscience
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Presynaptic CB1 Receptors Regulate Synaptic Plasticity at Cerebellar Parallel Fiber Synapses

2011

Endocannabinoids are potent regulators of synaptic strength. They are generally thought to modify neurotransmitter release through retrograde activation of presynaptic type 1 cannabinoid receptors (CB1Rs). In the cerebellar cortex, CB1Rs regulate several forms of synaptic plasticity at synapses onto Purkinje cells, including presynaptically expressed short-term plasticity and, somewhat paradoxically, a postsynaptic form of long-term depression (LTD). Here we have generated mice in which CB1Rs were selectively eliminated from cerebellar granule cells, whose axons form parallel fibers. We find that in these mice, endocannabinoid-dependent short-term plasticity is eliminated at parallel fiber…

PhysiologyPresynaptic TerminalsNeural facilitationNonsynaptic plasticityParallel fiberSynaptic TransmissionMice03 medical and health sciences0302 clinical medicineReceptor Cannabinoid CB1CerebellumMetaplasticitymedicineAnimalsLong-term depression030304 developmental biologyMice Knockout0303 health sciencesNeuronal PlasticitySynaptic scalingHomosynaptic plasticityChemistryLong-Term Synaptic DepressionGeneral NeuroscienceArticlesMice Inbred C57BLmedicine.anatomical_structurenervous systemSynaptic plasticityNeuroscience030217 neurology & neurosurgeryJournal of Neurophysiology
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A critical role for VEGF and VEGFR2 in NMDA receptor synaptic function and fear-related behavior

2016

Vascular endothelial growth factor (VEGF) is known to be required for the action of antidepressant therapies but its impact on brain synaptic function is poorly characterized. Using a combination of electrophysiological, single-molecule imaging and conditional transgenic approaches, we identified the molecular basis of the VEGF effect on synaptic transmission and plasticity. VEGF increases the postsynaptic responses mediated by the N-methyl-D-aspartate type of glutamate receptors (GluNRs) in hippocampal neurons. This is concurrent with the formation of new synapses and with the synaptic recruitment of GluNR expressing the GluN2B subunit (GluNR-2B). VEGF induces a rapid redistribution of Glu…

Vascular Endothelial Growth Factor A0301 basic medicine[SDV]Life Sciences [q-bio]Cell Culture TechniquesNonsynaptic plasticityBiologyNeurotransmissionHippocampusReceptors N-Methyl-D-AspartateSynaptic TransmissionMice03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicinePostsynaptic potentialAnimalsddc:610Molecular BiologyComputingMilieux_MISCELLANEOUSNeuronsNeuronal PlasticityBehavior AnimalGlutamate receptorExcitatory Postsynaptic PotentialsKinase insert domain receptorFearVascular Endothelial Growth Factor Receptor-2Protein SubunitsPsychiatry and Mental health030104 developmental biologySynaptic fatigueReceptors GlutamateSynapsesSynaptic plasticityNMDA receptorOriginal ArticleNeuroscience030217 neurology & neurosurgery
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